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Cell Death: Apoptosis

The procedure of programmed cell death, or apoptosis, is by and large portrayed by particular morphological qualities and vitality subordinate biochemical components. Cell death is a vital process of the cell to maintain the ratio of the cells such as cell movement. The cells of a multicellular organism are members of a highly organized community. The number of cells in this community is highly regulated and it done not only by controlling the rate of cell division, but also by controlling the rate of cell death. If cells are no longer needed, they destroy themselves by activating an intracellular death program. This process is called Apoptosis. The amount of apoptosis that occurs in developing and adult animal tissues can amaze us. If one will observe vertebrate nervous system, up to half of the nerve cells normally die soon after they are formed. In a healthy adult human, billions of cells die in the bone marrow and intestine every hour.

Related Journals of Cell Death: Apoptosis

Journal of Cell Science & Therapy, Journal of Stem Cell Research & Therapy, Cellular and Molecular Biology, Cell Biology: Research & Therapy, Cell, Journal of Cell Biology, Molecular Cell, Journal of Cell Science, Journal of Cellular Physiology, Neural Regeneration Research, Insights in Cell Science

Cell Death: Apoptosis

Expert PPTs

Speaker PPTs

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    Classically, the 3’untranslated region (3’UTR) is that region in eukaryotic protein-coding genes from the translation termination codon to the polyA signal. It is transcribed as an integral part of the mRNA encoded by the gene. However, there exists another kind of RNA, which consists of the 3’UTR alone, without all other elements in mRNA such as 5’UTR and coding region. The importance of independent 3’UTR RNA (referred as I3’UTR) was prompted by results of artificially introducing such RNA species into malignant mammalian cells. Since 1991, we found that the middle part of the 3’UTR of the human nuclear factor for interleukin-6 (NF-IL6) or C/EBP gene exerted tumor suppression effect in vivo. Our subsequent studies showed that transfection of C/EBP 3’UTR led to down-regulation of several genes favorable for malignancy and to up-regulation of some genes favorable for phenotypic reversion. Also, it was shown that the sequences near the termini of the C/EBP 3’UTR were important for its tumor suppression activity. Then, the C/EBP 3’UTR was found to directly inhibit the phosphorylation activity of protein kinase CPKC in SMMC-7721, a hepatocarcinoma cell line. Recently, an AU-rich region in the C/EBP 3’UTR was found also to be responsible for its tumor suppression. Recently we have also found evidence that the independent C/EBP 3’UTR RNA is actually exists in human tissues, such as fetal liver and heart, pregnant uterus, senescent fibroblasts etc. Through 1990’s to 2000’s, world scientists found several 3’UTR RNAs that functioned as artificial independent RNAs in cancer cells and resulted in tumor suppression. Interestingly, majority of genes for these RNAs have promoter-like structures in their 3’UTR regions, although the existence of their transcribed products as independent 3’UTR RNAs is still to be confirmed. Our studies indicate that the independent 3’UTR RNA is a novel non-coding RNA species whose function should be the regulation not of the expression of their original mRNA, but of some essential life activities of the cell as a whole.
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