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Cell Regeneration

 

Cell regeneration is the science in which the procedure of reestablishment, reclamation and development that makes genomes, cells, living beings and biological systems. Cell regeneration is a characteristic changes or occasions that cause aggravation or harm. Some parts of our bodies can repair themselves quite well after injury, but others don’t repair at all. We certainly can’t regrow a whole leg or arm, but some animals can regrow their body parts. Regeneration occurs in both human and animals. In Humans regrowth of a damaged organ part from the remaining tissue is known as regeneration. Humans are able to regenerate some organs, for example the liver. If a part of organ is lost by disease or injury, the organ grows back to its original size. Biggest example of regeneration in humans is our skin as it is constantly being renewed and repaired. Regeneration also occurs in animals. There are few animals that can regenerate their body parts. For example, flatworm or planarian can regenerate both the head from a tail piece, and the tail from a head piece.

Related Journals of Cellular and Molecular Biology

Journal of Cell Science & Therapy, Journal of Stem Cell Research & Therapy, Tissue Science & Engineering, , Cell, Journal of Cell Biology, Molecular Cell, Journal of Cell Science, Journal of Cellular Physiology, Neural Regeneration Research

Cell Regeneration

Expert PPTs

Speaker PPTs

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    Effect of liquiritigenin on apoptotic beta-cell death by palmitate-induced lipotoxicity in INS-1 cells
    | PDF Version
  • Dong-Kyu Kim
    Cross-Talk between Human Mast Cells and Epithelial Cells by IgE-mediated Periostin Production in Eosinophilic Nasal Polyps
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    The inflammasome of activated human B-cells regulates IL-1β and IgM: A crosstalk between the innate and the adaptive immune response
    | PDF Version
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    | PDF Version
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    | PDF Version
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    TRANSFORMING GROWTH FACTOR-β PROGRAMS CENTRAL-MEMORY DIFFERENTIATION IN EX-VIVO STIMULATED HUMAN T CELLS BY MODULATING ID3 EXPRESSION
    | PDF Version
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    Modulating the Antioxidant Defences to Potentiate Oncolytic Virotherapy in Refractory Cancer Cells
    | PDF Version
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    | PDF Version
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    | PDF Version
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    Microcalcification as an active phenomenon mediated by epithelial cells with mesenchymal characteristics
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    | PDF Version
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  • Yosef Yarden
    Classically, the 3’untranslated region (3’UTR) is that region in eukaryotic protein-coding genes from the translation termination codon to the polyA signal. It is transcribed as an integral part of the mRNA encoded by the gene. However, there exists another kind of RNA, which consists of the 3’UTR alone, without all other elements in mRNA such as 5’UTR and coding region. The importance of independent 3’UTR RNA (referred as I3’UTR) was prompted by results of artificially introducing such RNA species into malignant mammalian cells. Since 1991, we found that the middle part of the 3’UTR of the human nuclear factor for interleukin-6 (NF-IL6) or C/EBP gene exerted tumor suppression effect in vivo. Our subsequent studies showed that transfection of C/EBP 3’UTR led to down-regulation of several genes favorable for malignancy and to up-regulation of some genes favorable for phenotypic reversion. Also, it was shown that the sequences near the termini of the C/EBP 3’UTR were important for its tumor suppression activity. Then, the C/EBP 3’UTR was found to directly inhibit the phosphorylation activity of protein kinase CPKC in SMMC-7721, a hepatocarcinoma cell line. Recently, an AU-rich region in the C/EBP 3’UTR was found also to be responsible for its tumor suppression. Recently we have also found evidence that the independent C/EBP 3’UTR RNA is actually exists in human tissues, such as fetal liver and heart, pregnant uterus, senescent fibroblasts etc. Through 1990’s to 2000’s, world scientists found several 3’UTR RNAs that functioned as artificial independent RNAs in cancer cells and resulted in tumor suppression. Interestingly, majority of genes for these RNAs have promoter-like structures in their 3’UTR regions, although the existence of their transcribed products as independent 3’UTR RNAs is still to be confirmed. Our studies indicate that the independent 3’UTR RNA is a novel non-coding RNA species whose function should be the regulation not of the expression of their original mRNA, but of some essential life activities of the cell as a whole.
    | PDF Version
  • Fan-Gang Tseng
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    | PDF Version
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    PEGylated-thymoquinone-nanoparticle mediated retardation of breast cancer cell migration by deregulation of cytoskeletal actin polymerization through miR-34a
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    | PDF Version
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    | PDF Version
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