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Journal of Clinical & Experimental Neuroimmunology
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Dendritic Cell Immunology

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Dendritic Cell Immunology

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Dendritic Cell Immunology

It is also known as antigen presenting cells (accessory cells) of the mammalian immune system. It functions by processing antigen material and presenting it on to the cell surface of mainly T-cells of the immune system. They work as messengers between the innate and adaptive immune system.

Related Journals for Dendritic Cell Immunology

Journal of Clinical & Experimental Neuroimmunology, Journal of Neurological Disorders, Journal of Neurology & Neurophysiology, Journal of Neuroinfectious Diseases, Rheumatology: Current Research, Blue Books of Neurology, Egyptian Journal of Neurology, Psychiatry and Neurosurgery, Internet Journal of Neurology, Journal of Clinical Neurology, Romanian Journal of Neurology/ Revista Romana de Neurologie, CPD Bulletin Neurology, Current Clinical Neurology, Hot Topics in Neurology and Psychiatry

Dendritic Cell Immunology

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    The inflammasome of activated human B-cells regulates IL-1β and IgM: A crosstalk between the innate and the adaptive immune response
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    Make Your Immunology Research Easy
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    Longitudinal changes in airway microbiome signatures and immunoregulatory cell dynamics following Bronchial Thermoplasty
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  • Yosef Yarden
    Classically, the 3’untranslated region (3’UTR) is that region in eukaryotic protein-coding genes from the translation termination codon to the polyA signal. It is transcribed as an integral part of the mRNA encoded by the gene. However, there exists another kind of RNA, which consists of the 3’UTR alone, without all other elements in mRNA such as 5’UTR and coding region. The importance of independent 3’UTR RNA (referred as I3’UTR) was prompted by results of artificially introducing such RNA species into malignant mammalian cells. Since 1991, we found that the middle part of the 3’UTR of the human nuclear factor for interleukin-6 (NF-IL6) or C/EBP gene exerted tumor suppression effect in vivo. Our subsequent studies showed that transfection of C/EBP 3’UTR led to down-regulation of several genes favorable for malignancy and to up-regulation of some genes favorable for phenotypic reversion. Also, it was shown that the sequences near the termini of the C/EBP 3’UTR were important for its tumor suppression activity. Then, the C/EBP 3’UTR was found to directly inhibit the phosphorylation activity of protein kinase CPKC in SMMC-7721, a hepatocarcinoma cell line. Recently, an AU-rich region in the C/EBP 3’UTR was found also to be responsible for its tumor suppression. Recently we have also found evidence that the independent C/EBP 3’UTR RNA is actually exists in human tissues, such as fetal liver and heart, pregnant uterus, senescent fibroblasts etc. Through 1990’s to 2000’s, world scientists found several 3’UTR RNAs that functioned as artificial independent RNAs in cancer cells and resulted in tumor suppression. Interestingly, majority of genes for these RNAs have promoter-like structures in their 3’UTR regions, although the existence of their transcribed products as independent 3’UTR RNAs is still to be confirmed. Our studies indicate that the independent 3’UTR RNA is a novel non-coding RNA species whose function should be the regulation not of the expression of their original mRNA, but of some essential life activities of the cell as a whole.
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  • Fan-Gang Tseng
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    Engineered T cells: Next-generation cancer immunotherapy
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    Hematopoietic-stem-cell based therapy for HIV disease
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    AMNIOTIC DERIVED PROGENITOR CELLS IN DIFFERENT ANIMAL SPECIES IN VIEW OF CELL THERAPY APPLICATIONS
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    MEA-Based quantitative measurement of the electrophysiological activity of spiking cells
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    Mammalian cells communication a possible hypothesis
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    Global Metabolic Changes and Cellular Dysfunction in Diamide Challenged G6PD-Deficient Red Blood Cells
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    Brucellosis in cattle & buffaloes in and around Bikaner, Rajasthan, India
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