Case Report
Primate Erythroparvovirus 1 (Parvovirus B19): An Etiologic Agent of Ataxia and Cerebellar Disease?
Jacqueline A Hobbs1* and Hena Waseem21Department of Psychiatry and Pediatrics, University of Florida College of Medicine, USA
2Dartmouth Hitchcock Medical Center, USA
- *Corresponding Author:
- Jacqueline A Hobbs
Department of Psychiatry
University of Florida College of Medicine
Gainesville, Florida, USA
Tel: 352294-4945
Fax: 3525941818
E-mail: jahobbs@ufl.edu
Received date: Sep 19, 2016; Accepted date: Sep 29, 2016; Published date: Oct 03,2016
Citation: Hobbs JA, Waseem H (2016) Primate Erythroparvovirus 1 (Parvovirus B19): An Etiologic Agent of Ataxia and Cerebellar Disease. J Neuroinfect Dis 7: 227. doi:10.4172/2314-7326.1000227
Copyright: © 2016 Hobbs JA, et al. This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.
Abstract
Ataxia, a gross lack of motor control, is symptomatic of broader neurological disorder, typically of the cerebellum. A few case reports have documented the association of primate erythroparvovirus 1 [more commonly known as human parvovirus B19 (B19)] with ataxia. Parvoviruses are small DNA viruses that infect many different species. B19 is a well-known cause of erythema infectiosum, a common rash disease of childhood. B19 is also a cause of hydrops fetalis, a severe anemia of the fetus. Is it possible that the human parvovirus B19 is a cause of ataxia
